human insulin-specific elisa kit (Mercodia Inc)
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Human Insulin Specific Elisa Kit, supplied by Mercodia Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+insulin-specific+elisa+kit/mouse+insulin+elisa+kit/pm33906910-43-10-12
Average 90 stars, based on 1 article reviews
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other:Article Title: SEDDS for intestinal absorption of insulin: Application of Caco-2 and Caco-2/HT29 co-culture monolayers and intra-jejunal instillation in rats. Article Snippet: Accepted Manuscript SEDDS for intestinal absorption of insulin: Application of Caco-2 and Caco-2/ HT29 co-culture monolayers and intra-jejunal instillation in rats Jingying Liu, Ulrich Werner, Mario Funke, Melissa Besenius, Lasse Saaby, Mathias Fanø, Huiling Mu, Anette Müllertz PII: S0378-5173(19)30137-1 DOI: https://doi.org/10.1016/j.ijpharm.2019.02.014 Reference: IJP 18152 To appear in: International Journal of Pharmaceutics Received Date: 6 December 2018 Revised Date: 28 January 2019 Accepted Date: 12 February 2019 Please cite this article as: J. Liu, U. Werner, M. Funke, M. Besenius, L. Saaby, M. Fanø, H. Mu, A. Müllertz, SEDDS for intestinal absorption of insulin: Application of Caco-2 and Caco-2/HT29 co-culture monolayers and intra-jejunal instillation in rats, International Journal of Pharmaceutics (2019), doi: https://doi.org/10.1016/j.ijpharm.. 2019.02.014 This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Clinical Proteomics:Article Title: MEK/ERK Signaling in β-Cells Bifunctionally Regulates β-Cell Mass and Glucose-Stimulated Insulin Secretion Response to Maintain Glucose Homeostasis. Article Snippet: In diabetic pathology, insufficiency in b-cell mass, unable to meet peripheral insulin demand, and functional defects of individual b-cells in production of insulin are often concurrently observed, collectively causing hyperglycemia.. Here we show that the phosphorylation of ERK1/2 is significantly decreased in the islets of db/db mice as well as in those of a cohort of subjects with type 2 diabetes.. In mice with abrogation of ERK signaling in pancreatic b-cells through deletion of Mek1 and Mek2, glucose intolerance aggravates under high-fat diet– feeding conditions due to insufficient insulin production with lower b-cell proliferation and reduced b-cell mass, while in individual b-cells dampening of the number of insulin exocytosis events is observed, with the molecules involved in insulin exocytosis being less phosphorylated. Enzyme-linked Immunosorbent Assay:Article Title: MEK/ERK Signaling in β-Cells Bifunctionally Regulates β-Cell Mass and Glucose-Stimulated Insulin Secretion Response to Maintain Glucose Homeostasis. Article Snippet: In diabetic pathology, insufficiency in b-cell mass, unable to meet peripheral insulin demand, and functional defects of individual b-cells in production of insulin are often concurrently observed, collectively causing hyperglycemia.. Here we show that the phosphorylation of ERK1/2 is significantly decreased in the islets of db/db mice as well as in those of a cohort of subjects with type 2 diabetes.. In mice with abrogation of ERK signaling in pancreatic b-cells through deletion of Mek1 and Mek2, glucose intolerance aggravates under high-fat diet– feeding conditions due to insufficient insulin production with lower b-cell proliferation and reduced b-cell mass, while in individual b-cells dampening of the number of insulin exocytosis events is observed, with the molecules involved in insulin exocytosis being less phosphorylated. |
